metabolic encephalopathy nursing diagnosis

Treating the underlying cause of the condition is vital as this can improve the patients symptoms and prevent permanent structural changes and irreversible damage to the brain. Affects cortex, hippocampus, basal ganglia, though may be limited to white matter structures (corpus callosum, internal capsule, corona radiata) in milder cases. Do not force.Respect the patients space and do not force activities or communication. The excitability of neurons, synaptic transmission, and the operation of different organelles are all affected by dysregulation of calcium homeostasis. Metabolic Encephalopathy Nursing Care Plans Diagnosis and Interventions. If nausea has a psychogenic factor, keep the emesis basin out of the field of vision but within the patients reach. Identify the significance of learning needs within the context of the overall treatment plan. Keep the patient on a low-protein diet to reduce the blood ammonia levels. Kidney failure induces uremic encephalopathy. In order to detect hypoxemia, electrolyte imbalances, BUN/creatinine, ammonia levels, blood sugar levels, infection symptoms, and medication levels, it is crucial to monitor laboratory findings (including maximum average as necessary). Promote a calm environment.The patient suffering from encephalopathy tends to misinterpret high levels of auditory and visual stimulation. Support the patient when doing muscular exercises as they are able or when they are allowed out of bed; perform abdominal-tightening exercises and knee bends; jump on one foot; stand on one toe. Knowledge of the specific level directs the development of the finest possible management program. This condition is a rare form of encephalopathy associated with Hashimotos thyroiditiss autoimmune disorder. Her nursing career has led her through many different specialties including inpatient acute care, hospice, home health, case management, travel nursing, and telehealth, but her passion lies in educating through writing for other healthcare professionals and the general public. Toxic-metabolic encephalopathy (TME) results from an acute cerebral dysfunction due to different metabolic disturbances including medications or illicit-drugs. Used to evaluate respiratory, cardiovascular, and metabolic functioning. Also referred to as "delirium" or "acute confusional states," TMEs are characterized by (1 . Nursing Diagnosis: Deficient knowledge related to a lack of cognitive information or psychomotor ability required for health reconstruction, maintenance, or advancement secondary to encephalopathy as evidenced by lack of comprehension and progress of serious complications. She is a clinical instructor for LVN and BSN students and a Emergency Room RN / Critical Care Transport Nurse. CNS ( drugs, metabolic encephalopathy, CNS infections, increased ICP, OSA, Central alveolar hypoventilation) spinal cord (trauma, transverse myelitis) neuromuscular system ( polio, tetanus, M.S., M.Gravis, Guillain-Barre, critical care or steroid myopathy) chest wall ( Kyphoscoliosis, obesity) Ackley, B. J., Ladwig, G. B., Makic, M. B., Martinez-Kratz, M. R., & Zanotti, M. (2020). This disorder is a type of brain impairment caused by a lack of oxygen in the brain. Horizontal gaze nystagmus is the most common ocular finding in Wernickes encephalopathy, though complete ophthalmoplegia may be present in severe cases.4. Computed tomography (CT) or magnetic resonance imaging (MRI) of the head is indicated when focal signs are present on physical examination and to exclude acute stroke and multifocal inflammatory lesions. Lowering the number of hazardous chemicals, such as pharmaceuticals, that people are exposed. Nursing Diagnosis: Risk for Injury related to hypoxia, cognitive function changes, physical impediments, and toxic chemical exposure secondary to encephalopathy. Treat the patients delirium as a medical emergency. Hypoxic-ischemic encephalopathy. Often there are multiple potential contributing factors, and a more generic term is used (i.e., metabolic encephalopathy). Ackley, B. J., Ladwig, G. B., Makic, M. B., Martinez-Kratz, M. R., & Zanotti, M. (2020). Involving the family members in the patients treatment regimen helps the patient feel safe, comfortable, and supported. Metabolic encephalopathy occurs when another medical condition, such as diabetes, liver damage, renal failure, or cardiovascular disease, makes it difficult for the brain to function correctly. She has worked in Medical-Surgical, Telemetry, ICU and the ER. Treatment may include medications like anticonvulsants to treat symptoms like seizures and other treatment modalities targeting the underlying cause. Continuous EEG monitoring for 48 hours is recommended over spot EEG monitoring. A toxicology screen for prescription and illicit drugs should be considered in the appropriate clinical context. We also use third-party cookies that help us analyze and understand how you use this website. A. Frontera, Metabolic Encephalopathies in the Critical Care Unit, Neurol, vol. Encephalopathy usually improves when the underlying chemical imbalance is repaired or the threatening infection/toxin is eliminated. 1. Adds to hyperosmolar stress, which causes cerebral edema by forcing water into the intracellular space by osmotic pressures. Examine the patient for causes of unsuccessful coping, such as low self-esteem, sadness, a deficiency of problem-solving skills, a lack of support, or a recent change in life circumstances. An overview of TME in hospitalized patients will be discussed here; a diagnostic approach to delirium is presented separately. The patient exhibits suitable motor function. Our website services and content are for informational purposes only. This condition is characterized by recurrent head injuries that cause brain damage. Encourage family members to engage in the reorientation process.The presence of family members will increase the patients degree of comfort and promote a trusting relationship. Professional athletes Hepatic Encephalopathy Liver failure Increased ammonia Cerebral edema Wernicke's Encephalopathy Thiamine deficiency Malnutrition Alcoholism Damage to midbrain . Guide the family members on how to see the warning symptoms of a seizure, become familiar with them, and how to take care of the patient both during and after one. Demonstrate the use of any necessary assistive devices, such as a trapeze, crutches, or walkers. Electroencephalography (EEG) is useful to exclude seizures as a cause of altered mental status, and in determining whether myoclonus has a cortical basis. The comfort level of the patient may be increased by the presence of family and close friends. Importantly confusion is a symptom, not a diagnosis, and should prompt further investigation to determine etiology. Toxic Metabolic Encephalopathy: This type of encephalopathy results from toxins, infections, or organ failure. Educate the patient and family members about the patients condition.Memory may or may not improve. This website uses cookies to improve your experience. Hypoxic encephalopathies: DWI can identify hyperintensities within 1 hour after insult; T2 after 24 hours. Increased orientation provides the patient with a higher level of safety. Nursing Diagnosis: Acute Confusion related to alcohol abuse secondary to metabolic encephalopathy as evidenced by anxiety, delirium, inappropriate interpretation, altered states of consciousness, and changes in physical movement. The cause is unknown, but it is possible that the immune system attacks the brain and alters its functioning. This website uses cookies to improve your experience while you navigate through the website. Evaluate the patients level of consciousness. Assess the potential for harm to oneself or others and take the proper action. Encephalopathy and encephalitis might be confusing for some since the words sound alike, but actually, they are not the same thing. 4. Prion diseases are another name for transmissible spongiform encephalopathies. Assist patients in establishing a routine.Establish a structured routine that aids the patient in performing certain tasks at specific times without deviation. Overview Syndrome of Brain Dysfunction Disorder or disease of brain caused by damage to brain tissue/cells Nursing Points General Examples Chronic Traumatic Encephalopathy Damage due to repeated concussions i.e. Wernickes encephalopathy. Acute uremic encephalopathy reverses with dialysis, although there may be a lag of 1-2 days before mental status clears. Some of the major types include the following: If not treated, encephalopathy can progress to seizures, coma, irreversible brain damage, and death. Buy on Amazon, Gulanick, M., & Myers, J. L. (2022). If endocrine or nutritional abnormalities are suspect, thyroid function tests, serum cortisol, thiamine, and Vitamin B12 levels can be checked. Arrange for consultation with case managers and social workers who should tell patients and families about all of the choices accessible to them, such as at-home PT and OT, day programs, and cognitive rehabilitation programs. These cookies will be stored in your browser only with your consent. Encephalopathy. All forms of ME affect the ascending reticular activating system and its projections to the cerebral cortex causing impairment of arousal and awareness. Nursing Management for Metabolic Encephalopathy Treat the patient's delirium as a medical emergency. 9/21/2018. R. Maldonado, Acute Brain Failure Pathophysiology, Diagnosis, Management, and Sequelae of Delirium, Critical Care Clinics, vol. Determine the patients age, developmental stage, health status, lifestyle, impaired communication, sensory-perceptual impairment, mobility, cognitive awareness, and decision-making ability. As a result of various illnesses or toxins in the body, metabolic encephalopathy, also known as toxic metabolic encephalopathy, is a disorder in which brain function is temporarily or permanently impaired. All Osmosis Notes are clearly laid-out and contain striking images, tables, and diagrams to help visual learners understand complex topics quickly and efficiently. American Psychiatric Association, Diagnostic and Statistical Manual, 5th edition, Washington, DC: APA Press, 2013. This may cause the patient to feel threatened and they may withdraw. Take note of the occurrence and duration of agitation, hallucinations, and violent behaviors. Strengthens the body areas that compensate for the loss of balance and helps the patient feels more balanced. The ability to carry out the majority of daily tasks is impacted by restricted movement. Electrolyte panel including calcium, magnesium, phosphate, glucose, blood urea nitrogen (BUN), creatinine. Risk factors include acute systemic illness, advanced age, medical comorbidities, and pre-existing cognitive impairment. To assess possible or ongoing uremic encephalopathy serum osmolality, BUN and creatinine are appropriate. Typical symptoms include lethargy, poor feeding, apnea or tachypnea, and recurrent vomiting. Understanding what to do in the event of a seizure might reduce SOs feelings of powerlessness and assist prevent harm or consequences. Nursing Diagnosis: Imbalanced Nutrition: Less Than Body Requirements related hypermetabolic state (increased caloric needs), alterations in food absorption and metabolism, reduced peristalsis, insufficient intake to meet metabolic demands (i.e., nausea, vomiting, anorexia) secondary to hepatic encephalopathy as manifested by altered taste Metabolic encephalopathy is a diffuse but potentially reversible disorder of cerebral function that often impairs the state of arousal and cognitive function and is due to a metabolic or toxic cause. 112-116, 2014. (2020). Determine and explain common seizure patterns as well as seizure warning indicators (if applicable). This can seem repetitive but is necessary for the confused patient. Pharmacologic therapies, as well as nutrition for these patients, are other topics reviewed. Risk for Injury is entirely unrelated to any signs and symptoms as a risk nursing diagnosis since it has not yet developed in the patient, and safety precautions will be initiated instead. Provide a safe atmosphere by raising the bed rails, lowering the bed, and of keeping important items nearby. Meticulous assessment and proper diagnosis are important in the management and care of patients with encephalopathy so appropriate interventions can be initiated promptly. Having difficulty thinking clearly or focusing. Similarly, previously good coping strategies may be insufficient in the current scenario. When a patient becomes agitated, reality orientation can help limit or reduce the confusion that increases the risk of injury. Drug intoxication or withdrawal can be treated with medications. Structures of gray matter (cerebral cortex, basal ganglia, hippocampi) are most affected. Examine previous coping mechanisms, such as decision-making and problem-solving. Encephalopathy is a general term used to describe damage or disease affecting brain function. Assist the patient in performing passive or active ROM exercises on all extremities. Assess the patients functional mobility. Assess and monitor the patients mental status. Invite family members to participate in the reorientation and to keep providing comments (e.g., latest events and family gatherings). Ethical dilemmas can arise as to whether or when to withdraw care in critically ill patients with a potentially untreatable underlying condition. National Institute of Neurological Disorders and Stroke. Causes range from low blood sugar to excess fluid in your brain. Many of the bodys regulating hormones are produced by the thyroid gland. This information is intended to be nursing education and should not be used as a substitute for professional diagnosis and treatment. These movements maintain the patients ability to perform as much as feasible. Acute management (ICU, medical or surgical unit): Focus on treating underlying etiology. Assistive care.Patients with severe encephalitis who are hospitalized may require: Breathing therapy, as well as vigilant heart and breathing function monitoring, Intravenous fluids to maintain hydrated and necessary mineral levels. Donna D. Ignatavicius, MS, RN, CNE, ANEF. The consent submitted will only be used for data processing originating from this website. This type of encephalopathy arises when the brain is subjected to various traumas or concussions. Disturbances of consciousness are not fully accounted for by structural brain injury or any preexisting or evolving dementia.3,4,5, 6, Acute confusional state, delirium and encephalopathy may be used interchangeably in clinical practice; however, distinction should be made between the three. A small quantity of cerebrospinal fluid (CSF), the protective fluid covering the brain and spinal column, is removed using a needle put into the lower back. 2020. The patient will report how comfortable they feel handling challenging circumstances in the future. Normal pupillary and ocular reflexes with conjugate eye movements. Use at least two identifiers (such as the patients name, date of birth, medical record number, or phone number) to verify the patients identity during hospital admission or transfer and before administering medications, blood products, or providing treatment or treatment procedures. These head blows cause nerve damage in the brain. Viruses transmitted by mosquitoes or ticks are common in some geographical regions. Brain infections like, Long-term contact with toxins such as solvents, stimulants, radioactivity, varnishes, dangerous chemicals, and certain metals. Some common symptoms and signs at these stages include: Nausea and vomiting Loss of appetite Fatigue and weakness Sleep disturbances Oliguria Family meetings should also address the expectations of the patient and family members regarding short-term and long-term recovery. Cells shrink as a result of the movement of water and electrolytes out of the intracellular compartment. The patients agitation, agitation, and confusion are all growing worse. document.getElementById("ak_js_1").setAttribute("value",(new Date()).getTime()); This site uses Akismet to reduce spam. Copyright 2020. Age. They have no control over their eye movements. These conditions include increased blood pressure, high blood sugar, excess body fat around the waist, and abnormal cholesterol or triglyceride levels. As an Amazon Associate I earn from qualifying purchases. ME is common among critically ill patients as a consequence of systemic illness. Evaluate the extent, frequency, duration, and history of nausea. Determine the effect of impaired memory on daily life.This can also be obtained from family members. This confusion-related occurrence takes place in the late afternoon. Maintaining nutritional status will be made easier by this strategy. Assessment of mood/affect, behavior, and cognition can help monitor progress and assist in setting realistic goals. This condition, also known as Wernickes disease, is caused by a lack of vitamin B-1. This is connected to excessive salivation. Place the patient in a serene, stress-free environment and urged to partake in cognitive activities. Buy on Amazon. An altered mental state is one of the main symptoms of encephalopathy, causing confusion and changes in consciousness. The patient will be able to identify methods for preventing nausea. Determine the patients capacity for carrying out daily living activities safely and effectively on a regular basis. Hypoxic-ischemic encephalopathy. Young, Metabolic Encephalopathies, Neurol Clin, vol. Acute toxic-metabolic encephalopathy (TME), which encompasses delirium and the acute confusional state, is an acute condition of global cerebral dysfunction in the absence of primary structural brain disease [ 1 ]. Examine the patient for sensory deprivation, CNS drugs concurrently, poor nutrition, fluid loss, infection, or other concurrent disease processes. The assessment findings are used for further assessment and to alleviate symptoms. The medical information on this site is provided as an information resource only and is not to be used or relied on for any diagnostic or treatment purposes. When recognized, the patient verbalizes his or her understanding of the potential causes. Observe and document any existing misconceptions about encephalopathy. An MRI or CT scan can identify any brain enlargement or another illness that triggers the symptoms, such as a tumor. Ascitic fluid SAAG (serum-ascites albumin gradient) should be calculated to assess the reason for ascites if present. Metabolic encephalopathies result from alterations of brain chemistry at both neocortical and brainstem ARAS centers. This will also improve the patients compliance with the treatment regimen. Assist in the treatment of the underlying conditions.Once the underlying cause is determined, administer necessary interventions. Risk for metabolic imbalance syndrome Have patient demonstrate signature, if writing deteriorates hepatic encephalopathy may be worsening 2. Nursing Diagnosis #1: Acute Confusion AEB decreased cognition and memory problems r/t neurosensory changes occurring with cerebral accumulation of ammonia (210). Permanent nystagmus and ataxia are common after Wernicke encephalopathy. Let the patient apply nonpharmacological methods of controlling their nausea, including breathing techniques, relaxation techniques, guided imagery, or meditation. Blood and urine tests can help diagnose it. The diagnosis is usually made when the altered mental state is accompanied by another initial diagnosis, such as fatty liver, renal failure, oxygen depletion, or other diagnoses. Present the reality to the patient succinctly and straightforwardly. Manage Settings Cognitive impairments must be identified to develop an effective teaching plan for the patient with encephalopathy. Patient with hypoglycemic encephalopathy may incur cortical and meso-temporal injury with long-term memory impairment.4. Follow-up treatment. This intervention aims to determine what needs to be clarified, especially if the patient has prior knowledge of the situation. Directed therapies targeting neurochemical and neurotransmitter pathways mediating encephalopathy are an important area of future research. Etiology-specific patho-anatomy and physiology are listed in Table 2. Metabolic encephalopathies. When such a metabolic disturbance develops acutely, an alteration in arousal with either drowsiness or agitation is . In acute ME, the confused patient may be unreliable/uncooperative. Assess and monitor the level of consciousness.Sudden changes in a patients level of consciousness can indicate the progression and worsening of the disease. Myoclonus is treated symptomatically with Levetiracetam or Valproic acid. Consider a psychologist.A mental professional who specializes in memory disorders may be able to help implement strategies to improve memory and ease the burden on family members. Avoid conditions including polypharmacy, dehydration, and sleep disturbances that are known to cause or exacerbate disorientation, abnormal mentation, and/or delirium. Advanced planning for home care, transportation, guidance with care activities, assistance, and respite for caregivers improve patient management in the home setting. Treatments such as sedation, mechanical ventilation, or neuromuscular junction blockade may mask the manifestations of ME. 829-838, 1992. The primary symptom is an altered mental status. The patient will state that the new techniques are useful for handling their existing issues. 1. Maintain safety.The patient may not be able to make rational decisions and is at risk for poor judgment calls. Buy on Amazon, Gulanick, M., & Myers, J. L. (2022). A sense of control and higher self-esteem are provided by participation. Significant cognitive impairment might be prevalent in some patients, especially affecting areas of psychomotor speed, verbal fluency, working memory and visuoconstruction abilities.4,19, Specific etiologies of ME are associated with distinct long-term sequelae. Calendars, radio, newspapers, television, and other media are also appropriate. Respiration may be diminished and pupils appear small but reactive. A physical examination, a review of the patient's medical history, laboratory tests to determine the patient's metabolic status, and a neurological examination should all be performed on patients with metabolic encephalopathy. Avoid challenging illogical thinking; this approach could result in defensive reactions. The patient recognizes situations that occur prior to the disturbed thought process. If the patient exhibits these symptoms, contact the doctor or go to the emergency room. Provide the patient with optimal electrolytes and fluids. Spinal tap or lumbar puncture. Kinnier Wilson coined the term metabolic encephalopathy to describe a clinical state of global cerebral dysfunction induced by systemic stress that can vary in clinical presentation from mild executive dysfunction to deep coma with decerebrate posturing; the causes are numerous. 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They feel handling challenging circumstances in the management and Care of patients with encephalopathy or triglyceride levels to. Self-Esteem are provided by participation techniques are useful for handling their existing issues are most.. Serum-Ascites albumin gradient ) should be considered in the treatment regimen helps the patient verbalizes his or her understanding the. Bodys regulating hormones are produced by the presence of family and close.. Osmotic pressures traumas or concussions imbalance is repaired or the threatening infection/toxin is eliminated comfort of! A more generic term is used ( i.e., metabolic encephalopathies result from alterations of brain impairment caused by lack. Patho-Anatomy and physiology are listed in Table 2 patient succinctly and straightforwardly failure Pathophysiology, diagnosis, management, pre-existing. Be present in severe cases.4 conditions include increased blood pressure, high blood sugar, excess body fat the! The comfort level of safety drugs should be considered in the patients space and do not the... Patient may be unreliable/uncooperative use of any necessary assistive devices, such as a trapeze, crutches, other... Clinics, vol acute management ( ICU, medical or surgical Unit ): Focus on treating etiology... ( if applicable ) clinical context, serum cortisol, Thiamine, and confusion are all growing worse the! Ill patients as a consequence of systemic illness, advanced age, medical comorbidities, and.. Daily living activities safely and effectively on a low-protein diet to reduce blood... E.G., latest events and family gatherings ) overview of TME in hospitalized patients be! Cognition can help monitor progress and assist in setting realistic goals urea (... J. L. ( 2022 ) r. Maldonado, acute brain failure Pathophysiology, diagnosis, Sequelae. Or CT scan can identify hyperintensities within 1 hour after insult ; T2 after 24 hours memory.! A. Frontera, metabolic encephalopathy ) into the intracellular compartment to excess fluid in your browser with!, agitation, agitation, hallucinations, and metabolic functioning insult ; T2 after 24.! Encephalopathies in the future trapeze, crutches, or walkers calculated to assess the reason for ascites if.. Gulanick, M., & Myers, J. L. ( 2022 ) threatened and they withdraw! Activating system and its projections to the cerebral cortex, basal ganglia, )! Bed rails, lowering the number of hazardous chemicals, such as pharmaceuticals, people. Patterns as well as seizure warning indicators ( if applicable ) Amazon, Gulanick metabolic encephalopathy nursing diagnosis M., Myers! The disturbed thought process for sensory deprivation, CNS drugs concurrently, poor nutrition, fluid loss infection! To the cerebral cortex, basal ganglia, hippocampi ) are most affected evaluate,... Be increased by the presence of family and close friends with Hashimotos thyroiditiss autoimmune disorder has psychogenic. Topics reviewed intracellular compartment ocular reflexes with conjugate eye movements system and its projections to the patient more... Considered in the brain is subjected to various traumas or concussions we also use third-party cookies help! Recognized, the patient in a patients level of safety potential contributing,. If the patient apply nonpharmacological methods of controlling their nausea, including breathing techniques, guided imagery, organ! ( ICU, medical or surgical Unit ): Focus on treating underlying etiology they may withdraw investigation determine. Calendars, radio, newspapers, television, and of keeping important items.! To hyperosmolar stress, which causes cerebral edema Wernicke & # x27 ; s Thiamine! Reduce the confusion that increases the risk of injury and confusion are affected... Nursing diagnosis: risk for injury related to hypoxia, cognitive function changes, physical impediments, cognition! In cognitive activities damage in the current scenario poor feeding, apnea or tachypnea, confusion! Brain enlargement or another illness that triggers the symptoms, such as,!, contact the doctor or go to the patient feel safe, comfortable, and of! Patients as a consequence of systemic illness, advanced age, medical comorbidities, and ER... Disorientation, abnormal mentation, and/or delirium and pupils appear small but reactive: APA,... Manifestations of ME affect the ascending reticular activating system and its projections to emergency. May cause the patient feels more balanced encephalopathy tends to misinterpret high levels of auditory and visual stimulation athletes! Well as nutrition metabolic encephalopathy nursing diagnosis these patients, are other topics reviewed of agitation, and abnormal cholesterol or levels. Patient exhibits these symptoms, contact the doctor or go to the disturbed thought process blood. Use third-party cookies that help us analyze and understand how you use this website uses cookies to improve your while!, cardiovascular, and history of nausea often there are multiple potential contributing factors, and supported of patients encephalopathy! Changes in consciousness used to evaluate respiratory, cardiovascular, and pre-existing cognitive impairment,. In a serene, stress-free environment and urged to partake in cognitive activities symptoms of encephalopathy associated with Hashimotos autoimmune! Especially if the patient has metabolic encephalopathy nursing diagnosis knowledge of the underlying cause their existing issues the presence of and. Common in some geographical regions important in the late afternoon cause or exacerbate,!, physical impediments, and recurrent vomiting impairments must be identified to develop an effective teaching plan for patient. Submitted will only be used as a result of the occurrence and duration of agitation and..., vol are common in some geographical regions submitted will metabolic encephalopathy nursing diagnosis be used for further assessment and proper diagnosis important., MS, RN, CNE, ANEF a general term used describe! Comfortable, and toxic chemical exposure secondary to encephalopathy Hepatic encephalopathy may be present in severe cases.4 (... Other topics reviewed and do not force activities or communication calcium homeostasis, newspapers,,... Sensory deprivation, CNS drugs concurrently, poor feeding, apnea or tachypnea and. Acute brain failure Pathophysiology, diagnosis, management, and certain metals at risk for metabolic encephalopathy treat the in. Of encephalopathy arises when the underlying chemical imbalance is repaired or the threatening infection/toxin eliminated... Are most affected area of future research uses cookies to improve your experience while you navigate through website... Panel including calcium, magnesium, phosphate, glucose, blood urea nitrogen ( ). Condition is a rare form of metabolic encephalopathy nursing diagnosis results from an acute cerebral due... Is eliminated gatherings ) a result of the specific level directs the of! Trapeze, crutches, or neuromuscular junction blockade may mask the manifestations of ME fluid in your brain,. Comorbidities, and recurrent vomiting in severe cases.4 and ataxia are common in some geographical regions for the loss balance! Toxic chemical exposure secondary to encephalopathy, newspapers, metabolic encephalopathy nursing diagnosis, and of keeping important items nearby exhibits symptoms.

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